Complete list of Blogger account limits

by Greenlava

Blogger is a free blogging service hosted by Google. Here is some information about the limits a Blogger account has. I’ve put them into a list for your reference. I’m sure this information will come in handy at some point in your blogging future. So you might want to bookmark this post, just in case.

Account limits

• Number of blogs: 100 per account.
• Profile "About Me" info length: 1,200 characters.
• Profile Interests and Favorites: 2,000 characters in each field.
• Number of blogs to follow: 300 blogs per account.
• Image size (dimension)
  • Image uploaded via post editor: 1600px wide or high. Larger images can be uploaded but it will be scaled down to 1600px while preserving the aspect ratio
  • Image (for background) uploaded via Template > Customize > Background: No limit
• Image filesize (byte)
  • Image uploaded via post editor: 8MB
  • Image (for background) uploaded via Template > Customize > Background: 300kB
  • Uploaded via Blogger Mobile 250KB
• Capacity of image and video hosting (on Blogger partner Picasa Web Albums):
  • Photos up to 2048 x 2048 pixels and videos up to 15 minutes: No limit and free
  • Larger photos and videos: 1024MB free. Need to buy for more storage.
• Bandwidth of image hosting: No limit

Blog limits

• Length of blog title: 90 characters.
• Length of subdomain name (as in http://SUBDOMAIN.blogspot.com): 37 characters.
• Length of blog description: 500 characters.
• Number of members/authors: 100 member invitations per blog.
• Number of readers (for a private blog): 100 per blog.
• Number of posts: No limit.
• Number of posts displayed on multiple-post pages (homepage, archive etc.):
  Blogger will attempt to display the number of posts as set in Settings > Posts and Comments > Show At Most provided the posts’ file size does not exceed the Blogger auto-pagination cap of 1MB.
  Bigger posts may cause fewer number of posts to be displayed.
• Number of static pages: 20 per blog (updated Mar 2012).
• Number of labels: 5000 unique labels per blog.

Post limits

• Length of individual post: No limit.
• Size of individual post: No limit, but very large posts may run you up against the multiple-post page size limit (see above).
• Number of labels: 20 unique labels per post. All labels combined must not exceed 200 characters per post (thanks to Zipote Craz).
• Number of comments: No limit.
• Length of post filename: 39 characters (auto-generated from post title, truncated to length).

Other limits

• Comment length: 4,096 characters per comment.

I hope you find the information useful. If you know a limit that’s not in the list, please share them in the comments.

Enjoy!

http://www.bloggersentral.com/2011/03/limits-on-blogger-account.html

Extracts ....

Do You Want to Radically Improve Your Health?


Then replace soda and other sugary drinks with clean, pure water.

Normalizing your insulin levels is one of the most powerful physical actions you can take to improve your health and lower your risk of cancer along with all the other diseases and long-term chronic health conditions mentioned above.

Fortunately, it is also the variable most easily influenced by healthy eating and exercise.

Dr Mercola: http://healthticket.blogspot.com/2012/09/10-diseases-linked-to-soda.html

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With all of the attention surrounding diabetes these days, you’ve probably heard of a condition called metabolic syndrome—a collection of risk factors that are linked to type 2 diabetes, cardiovascular disease and stroke. These risk factors include high blood pressure, high triglycerides, low HDL (“good”) cholesterol, raised fasting glucose levels and a waist circumference measurement of 35 inches or more (think “beer gut”). If you have three of these five risk factors, then you may have metabolic syndrome, which puts you at increased risk of more serious complications.

Insulin resistance is the cornerstone of metabolic syndrome. Your pancreas produces the hormone insulin, which is necessary for the absorption and utilization of sugar in your diet. However, when you have insulin resistance, your body doesn’t respond as accurately to insulin, which causes the pancreas to produce more insulin.

This is where the merry-go-round starts, because the more insulin you have in the bloodstream, the greater your risk for type 2 diabetes.

Along with insulin resistance, endothelial dysfunction is a hallmark of metabolic syndrome. Endothelial dysfunction is characterized by narrowed blood vessels, inflammation and pro-thrombotic properties. In fact, it is one of the first signs of atherosclerosis and impending cardiovascular disease.

When the endothelial lining becomes damaged, NO production suffers, making all the blood vessels susceptible to inflammation, clotting and other negative effects.

While there’s little doubt that antioxidants play an important part in protecting the heart, a team of researchers recently examined the effect of one particular type of antioxidant—grape polyphenols—on the reduction of metabolic syndrome risk factors, especially the cardiovascular ones.²
Grapes contain numerous antioxidant polyphenols—anthocyanins, flavonols and resveratrol, to name a few. Resveratrol, in particular, has been shown to be extremely heart healthy by reducing LDL cholesterol and inflammation.^3-4 As many wine lovers know, red wine is high in resveratrol, and when consumed in moderation (one to two glasses per day), has some impressive heart-protective properties.

Go for the GrapeBecause metabolic syndrome has so many different factors, no simple pharmaceutical solutions exist to treat it. The best solution doctors can offer is to adjust certain lifestyle factors like diet and exercise. This, of course, is sound advice.

Everyone should be eating a clean diet and exercising anyway, but those with metabolic syndrome could see dramatic changes in their condition with these two changes alone. However, considering the proven health benefits of grape polyphenols, it’s great news indeed that this nutrient can now be added to your arsenal in the war against metabolic syndrome and its health effects.

A good way to make grape polyphenols a part of your daily regimen—particularly if you have metabolic syndrome or if you are at risk of developing cardiovascular disease—is to simply make red grapes a regular part of your diet. Eat them as is, or add them to salads and smoothies. And, as mentioned earlier, red wine is a good option to boost your polyphenol intake. Just be sure not to overindulge—drink no more than one or two glasses a day.

However, to achieve real therapeutic benefit, you may want to think about taking grape polyphenols in supplement form. Fortunately, polyphenols like resveratrol and grape seed extract are readily available at most health food stores and vitamin retailers.

http://healthticket.blogspot.com/2012/12/grape-polyphenols-reduce-metabolic.html

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Increased blood level of homocysteine is a strong risk factor for the development of Alzheimer disease and dementia31. Three B vitamins, folic acid, B6, and B12, can help lower your homocysteine levels. Fortified cereal, other grains, and leafy green vegetables are good sources of B vitamins.

eMedExpert.com  http://healthticket.blogspot.com/2012/09/how-to-boost-brain-power.html

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Omega-3 and Turmeric

The results from the third diet (Omega-3 and turmeric) were astounding. DHA has been shown in other studies to repair cell membrane damage but scientists discovered that the healing capabilities of omega-3 fatty acids include the ability to protect nerves after an accident or injury and to prevent cell death.

This total cellular protection is crucial to successful nerve injury recovery.

Curcumin’s role in recovery includes relieving and reducing inflammation, thereby preventing further nerve damage and speeding recovery. Curcumin is such a potent antioxidant that it has been shown to rebuild brain cells and aid in stroke prevention.

http://healthticket.blogspot.com/2012/10/spinal-cord-injury-omega-3-and-turmeric.html

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Vitamin K2 and Natto

In the meantime, since nearly 100 percent of people don't get sufficient amounts of vitamin K2 from their diet to reap its health benefits, you can assume you need to bump up your vitamin K2 levels by modifying your diet or taking a high-quality supplement.

As for dietary sources, eating lots of green vegetables, especially kale, spinach, collard greens, broccoli, and Brussels sprouts, will increase your vitamin K1 levels naturally. For vitamin K2, cheese and especially cheese curd is an excellent source. The starter ferment for both regular cheese and curd cheese contains bacteria -- lactococci and proprionic acids bacteria -- which both produce K2.

You can also obtain all the K2 you'll need (about 200 micrograms) by eating 15 grams of natto daily, which is half an ounce. It's a small amount and very inexpensive, but many Westerners do not enjoy the taste and texture.

If you don't care for the taste of natto, the next best thing is a high-quality K2 supplement. Remember you must always take your vitamin K supplement with fat since it is fat-soluble and won't be absorbed without it.

Although the exact dosing is yet to be determined, Dr. Vermeer recommends between 45 mcg and 185 mcg daily for adults. You must use caution on the higher doses if you take anticoagulants, but if you are generally healthy and not on these types of medications, I suggest 150 mcg daily.

http://healthticket.blogspot.com/2011/11/importance-of-vitamin-k.html

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These natural therapies knocked the cancer out

The doctor cured the boy's tumors with a combination of natural therapies, including:

• Hyperthermia: a gentle therapy that gives the patient an artificial fever to "bake" the cancer cells to death. Cancer cells "can't take the heat," but normal cells can.
• Oxygen therapy: Cancer cells die by the millions when they get a blast of oxygen.
• Laetrile and vitamin C by IV: These non-toxic therapies kill cancer cells and also quench free radicals. They are far more powerful when administered by IV than when taken by mouth.
• A strictly organic diet to give the body the nutrition it needs
• Detoxification therapies to get rid of the poisons that can cause cancer

Source:  The Amish Cancer Secret

http://www.cancerdefeated.com/amish-secret/index.php?

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Today, we know its cancer-curing effects are from its numerous anti-cancer and antioxidant compounds. Cabbage speeds up estrogen metabolism, which is thought to help block breast cancer and suppress growth of polyps, a prelude to colon cancer. According to research, eating cabbage more than once a week cut men's colon cancer odds by as much as 66 percent. As little as two daily tablespoons of cooked cabbage protected subjects against stomach cancer. Cabbage also contains powerful anti-ulcer compounds; its juice has shown to help heal ulcers in humans.
- Timeless Secrets of Health & Rejuvenation: Unleash The Natural Healing Power That Lies Dormant Within You by Andreas Moritz

http://healthticket.blogspot.com/2012/12/stop-stomach-ulcers-with-miracle-remedy.html

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 Supercharge Your Health With Seven Medicinal Herbs & Spices

Ginger – Over 50 antioxidants have been found in ginger. It helps increase circulation, calms digestive problems. Ginger has also been used to treat food poisoning, shown to lower cholesterol, treat arthritis, reduce inflammation, and can be used to help increase insulin sensivity in diabetics.

http://healthticket.blogspot.com/2013/08/supercharge-your-health-with-seven.html

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The research team was highly impressed with their results. Those who took the pomegranate extract showed a significant decrease in a specific, important marker linked to cell damage.

That marker, known as 8-Oxo-DG, can have detrimental effects such as:

• Lessened brain function
• Weakening of muscles
• Impaired kidney function
• Decrease in liver productivity
• Aging of the skin

Sergio Streitenberger, lead researcher at ProbelteBio, believes the study “demonstrates that the regular consumption of this pomegranate extract can slow down the process of DNA oxidation.”

The pomegranate’s ability to decrease 8-Oxo-DG suggests that it wields significant power as an anti-aging super food.

Pomegranate Benefits The Elixir of Youth Hidden in...

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Curcumin Starves Cancer Cells to Death - by Life Extension

Cancer cells require tremendous energy to promote their rapid growth. A new study shows how curcumin can selectively starve tumor cells.

Winter 2018-2019 Special Edition

By Kirk Stokel

(Previously published here on 7 October 2019)

Over 4,500 published studies describe the anti-cancer effects of curcumin.

Researchers are intrigued by a promising discovery that curcumin selectively starves tumor cells to death.1

Curcumin does this by depriving cancer cells of the ability to make and use ATP, the energy currency within cells.2

Since most cancer cells generate ATP in a different way than healthy cells, curcumin selectively kills tumor cells with no impact on healthy tissues.

These newly uncovered energy-disrupting properties of curcumin further support the preventive potential of this botanical compound.

Cutting Cancer’s Energy Supply

Cancer cells require tremendous energy to promote their rapid, uncontrolled growth, and they have developed numerous methods to support that energy demand.

In particular, cancer cells have an unusual, oxygen-free mechanism of extracting energy from glucose to produce ATP (adenosine triphosphate). This process helps most cancer cells survive in the low-oxygen environments they generate as they grow rapidly.2

Scientists conducted a study to determine if this process can be reversed in cancer cells.

Since many different types of cancer use the same metabolic trick to extract energy, the researchers studied curcumin’s impact on a variety of tumor lines, including leukemia, breast, melanoma (skin), and colon cancers.2

This study shed new light on curcumin’s ability to starve cancer into submission by interfering with how tumors cells use energy.

Let’s look at the findings one at a time.

Curcumin Blocks ATP Production

First, the researchers found that curcumin can sharply reduce how much energy is available to the cancer cells.2

This study showed that curcumin was able to:

1) Reduce levels of ATP-synthase (the enzyme that creates ATP) in all four tumor cell lines in culture,

2) Reduce cellular levels of ATP in three of the four cell lines, and

3) Lower the ratio of high-energy ATP to lower-energy AMP in all four cell lines.

Curcumin Slows Tumor Growth

Next, researchers implanted aggressive skin cancer (melanoma) cells into live mice. Half the mice were treated with curcumin and the other half served as the control group.2

Just 2 days into the study, the curcumin-treated mice were demonstrating significantly slower tumor growth. Tumor growth remained significantly slower for the entire period of the study.

Not surprisingly, ATP-synthase, ATP levels, and the ATP/AMP ratio were all significantly lowered in the curcumin group. These results indicate that energy starvation was a mechanism of action by which curcumin slowed tumor growth.

Curcumin Prevents New Blood Vessel Growth

Cancers need to trigger new blood vessel development (called angiogenesis) to support their nutrient needs as they rapidly grow. Blocking this process is an important way to limit the growth and spread of a tumor.

After removing tumors from the mice in this study, the researchers found that the curcumin-supplemented animals’ tumors had fewer new blood vessels compared with control-fed mice.

This indicates that curcumin reduced the tumors nutrient energy access by limiting blood flow.2

Curcumin’s Known Tumor-Fighting Properties

This most recent study adds to an abundance of evidence pointing to curcumin’s potent cancer-fighting abilities. It focuses on curcumin’s unique ability to combat a variety of cancers by cutting off their energy supply.

Previous studies have shown curcumin’s tumor-fighting properties in multiple specific types of cancer. Here are some highlights from the most recent literature.

Colorectal Cancer

Cancers of the colon and rectum are among the most common malignancies in men and women. Curcumin has a long track record of achievements in preventing colorectal cancers.

A Phase IIa human clinical trial has shown that 4 grams/day of curcumin significantly reduces the number of aberrant crypt foci found during endoscopy. This is a critical finding because if aberrant crypt foci are left untreated, they can produce malignant tumors.3

Weight loss is common in cancer patients. A human study showed that colon cancer patients who were supplemented with curcumin gained more weight, had less inflammation, and had increased numbers of cancer cells dying by apoptosis.4 This effect has been traced to curcumin’s ability to activate a “programmed cell death” gene in tumor cells.

Finally, curcumin has been shown to selectively reduce the survival of cancer stem cells.5 These cells account for much of the metastatic spread and tumor recurrences seen in aggressive malignancies. Cutting their numbers is an appealing approach to preventing tumors from spreading.

While a high-dose (4,000 mg) of curcumin was used in this study, more bioavailable curcumin forms can enable one to achieve a similar dose with two capsules a day.

WHAT YOU NEED TO KNOW

Curcumin Starves Cancer Cells to Death

• Curcumin is a powerful, multi-functional polyphenol that is gaining increasing recognition for its cancer chemopreventive properties.
• A new study shows that curcumin sharply restricts cancer cells’ ability to extract energy from glucose in the blood.
• This mechanism is especially appealing for cancer chemoprevention because it would target the great majority of cancers.
• Previous studies also show that curcumin can reduce inflammation, prevent chemical stress, shut down cancer-promoting pathways, and interfere with malignant cells’ growth and development.
• These multiple mechanisms of action make it clear that curcumin should be a central element of any comprehensive cancer chemoprevention regimen.

Breast Cancer

Breast cancer is the most common cancer in women, and it’s the second leading cause of cancer deaths among women worldwide.6,7

Curcumin has shown promise in combatting breast cancer. This is especially true of estrogen receptor-negative tumors, which respond poorly to cancer chemotherapy.6

Studies show that giving curcumin to animals with implanted human breast cancers shrinks the tumors, deprives them of their blood supply, and triggers their self-destruction by apoptosis.6,8

More recently, research revealed that curcumin can also combat breast cancer by reversing the excessive methylation of certain genes that is associated with increased cancer development.9

Prostate Cancer

Prostate cancer strikes one in nine American men, yet is among the most preventable cancers because of its slow growth.10

Curcumin has multiple actions against prostate cancer. It defends prostate cells against the dysfunctional proteins produced during cancer progression. It also slows invasion of nearby healthy tissue by the cancer, which helps to keep the tumor at a lower grade (meaning it grows more slowly and has a better prognosis).11-13

One study showed that when human prostate cells were implanted into mice, they grew into sizable tumors. But when these animals were supplemented with curcumin, the tumors grew 27% more slowly. In addition, the time it took to double levels of prostate specific antigen (PSA) was extended or delayed by about two-fold.14 (Doubling time is an indicator of how aggressive the cancer is.)

Curcumin can also reduce tumor-derived testosterone production in prostate cancer cells.15 This is a hidden source of male hormones that often contributes to treatment-resistant disease.16 This development offers a welcome new approach to treating these challenging tumors.

Liver Cancer

Liver cancer is increasing in incidence worldwide, spurred by epidemic hepatitis C virus and rampant fatty liver disease.17 

In lab studies, curcumin was shown to help prevent liver cancer by reducing inflammatory signaling, inhibiting cell growth, and activating cell death by apoptosis.18-20 It has also been shown to reduce populations of liver cancer stem cells.19,21

In one study, curcumin inhibited the growth of liver cancer cells, while also promoting their death by apoptosis. And in a mouse model of liver cancer, treatment with curcumin caused the tumors to grow much more slowly.22

And, in an exciting development, a study published in 2018 showed that when curcumin is combined with the AMPK-activator drug metformin, the combination worked better than either alone in preventing growth, metastasis, and new blood vessel formation in hepatocellular carcinoma, the most common—and most deadly—type of liver cancer.23

Lung Cancer

A new study shows that
curcumin sharply restricts
cancer cells’ ability to
extract energy from
glucose in the blood.

Lung cancer continues to be the top cause of cancer-related deaths, making it a huge priority for prevention.24,25

An abundance of animal and basic lab studies show curcumin’s potential against lung cancer.

For example, curcumin has been shown to reduce the growth of implanted human lung cancers in mice.26  Lab studies on lung cancer show that curcumin alters proteins required for metastasis, boosting the function of immune cells that are inactivated by cancers, and targeting blood vessel growth.24,26,27

A unique way in which curcumin combats lung cancer is by modulating microRNA inside cancer cells.28-30 These short stretches of genetic material regulate how the main genes in tumor cells are translated into functional proteins.

Studies reveal that curcumin inhibits lung cancer cell growth by downregulating a gene that promotes tumor formation, while upregulating genes that suppress transformation.28 Other research shows similar favorable modulation of miRNAs involved in metastatic spread.30

Summary

Curcumin is the polyphenol that gives the yellow color to the spice turmeric. It has been front and center in the scientific press for years—mostly recognized for its ability to suppress inflammation.2,31,32

Curcumin has also been shown to prevent cancer progression through a variety of mechanisms. Most recently, a study found that curcumin starves cancer cells of much-needed energy.

This mechanism is especially appealing for cancer prevention because it would target the great majority of cancers.

This confirms previous studies showing that curcumin has specific effects against colorectal, breast, prostate, lung, and liver cancers.

Starving malignant cells of their energy supply is an ideal way of boosting our bodies’ natural cancer resistance, helping it quench cancer long before a tumor is detectable.

If you have any questions on the scientific content of this article, please call a Life Extension® Wellness Specialist at 1-866-864-3027.

References

1. Available at: https://www.ncbi.nlm.nih.gov/pubmed/?term=curcumin+and+cancer. Accessed September 21, 2018.
2. Bianchi G, Ravera S, Traverso C, et al. Curcumin induces a fatal energetic impairment in tumor cells in vitro and in vivo by inhibiting ATP-synthase activity. Carcinogenesis. 2018 Sep 21;39(9):1141-50.
3. Carroll RE, Benya RV, Turgeon DK, et al. Phase IIa clinical trial of curcumin for the prevention of colorectal neoplasia. Cancer Prev Res (Phila). 2011 Mar;4(3):354-64.
4. He ZY, Shi CB, Wen H, et al. Upregulation of p53 expression in patients with colorectal cancer by administration of curcumin. Cancer Invest. 2011 Mar;29(3):208-13.
5. James MI, Iwuji C, Irving G, et al. Curcumin inhibits cancer stem cell phenotypes in ex vivo models of colorectal liver metastases, and is clinically safe and tolerable in combination with FOLFOX chemotherapy. Cancer Lett. 2015 Aug 10;364(2):135-41.
6. Bimonte S, Barbieri A, Palma G, et al. Dissecting the role of curcumin in tumour growth and angiogenesis in mouse model of human breast cancer. Biomed Res Int. 2015;2015:878134.
7. Wang Y, Yu J, Cui R, et al. Curcumin in Treating Breast Cancer: A Review. J Lab Autom. 2016 Dec;21(6):723-31.
8. Ferreira LC, Arbab AS, Jardim-Perassi BV, et al. Effect of Curcumin on Pro-angiogenic Factors in the Xenograft Model of Breast Cancer. Anticancer Agents Med Chem. 2015;15(10):1285-96.
9. Kumar U, Sharma U, Rathi G. Reversal of hypermethylation and reactivation of glutathione S-transferase pi 1 gene by curcumin in breast cancer cell line. Tumour Biol. 2017 Feb;39(2):1010428317692258.
10. Available at: https://www.cancer.org/cancer/prostate-cancer/about/key-statistics.html. Accessed September 24, 2018.
11. Sundram V, Chauhan SC, Ebeling M, et al. Curcumin attenuates beta-catenin signaling in prostate cancer cells through activation of protein kinase D1. PLoS One. 2012;7(4):e35368.
12. Liu T, Chi H, Chen J, et al. Curcumin suppresses proliferation and in vitro invasion of human prostate cancer stem cells by ceRNA effect of miR-145 and lncRNA-ROR. Gene. 2017 Oct 5;631:29-38.
13. Yang J, Wang C, Zhang Z, et al. Curcumin inhibits the survival and metastasis of prostate cancer cells via the Notch-1 signaling pathway. APMIS. 2017 Feb;125(2):134-40.
14. Hong JH, Lee G, Choi HY. Effect of curcumin on the interaction between androgen receptor and Wnt/beta-catenin in LNCaP xenografts. Korean J Urol. 2015 Sep;56(9):656-65.
15. Ide H, Lu Y, Noguchi T, et al. Modulation of AKR1C2 by curcumin decreases testosterone production in prostate cancer. Cancer Sci. 2018 Apr;109(4):1230-8.
16. Armandari I, Hamid AR, Verhaegh G, et al. Intratumoral steroidogenesis in castration-resistant prostate cancer: a target for therapy. Prostate Int. 2014 Sep;2(3):105-13.
17. Global Burden of Disease Liver Cancer C, Akinyemiju T, Abera S, et al. The Burden of Primary Liver Cancer and Underlying Etiologies From 1990 to 2015 at the Global, Regional, and National Level: Results From the Global Burden of Disease Study 2015. JAMA Oncol. 2017 Dec 1;3(12):1683-91.
18. Dai XZ, Yin HT, Sun LF, et al. Potential therapeutic efficacy of curcumin in liver cancer. Asian Pac J Cancer Prev. 2013;14(6):3855-9.
19. Marquardt JU, Gomez-Quiroz L, Arreguin Camacho LO, et al. Curcumin effectively inhibits oncogenic NF-kappaB signaling and restrains stemness features in liver cancer. J Hepatol. 2015 Sep;63(3):661-9.
20. Elmansi AM, El-Karef AA, Shishtawy M, et al. Hepatoprotective Effect of Curcumin on Hepatocellular Carcinoma Through Autophagic and Apoptic Pathways. Ann Hepatol. 2017 Jul-Aug;16(4):607-18.
21. Tsai CF, Hsieh TH, Lee JN, et al. Curcumin Suppresses Phthalate-Induced Metastasis and the Proportion of Cancer Stem Cell (CSC)-like Cells via the Inhibition of AhR/ERK/SK1 Signaling in Hepatocellular Carcinoma. J Agric Food Chem. 2015 Dec 9;63(48):10388-98.
22. Pan Z, Zhuang J, Ji C, et al. Curcumin inhibits hepatocellular carcinoma growth by targeting VEGF expression. Oncol Lett. 2018 Apr;15(4):4821-6.
23. Zhang HH, Zhang Y, Cheng YN, et al. Metformin incombination with curcumin inhibits the growth, metastasis, and angiogenesis of hepatocellular carcinoma in vitro and in vivo. Mol Carcinog. 2018 Jan;57(1):44-56.
24. Liu D, You M, Xu Y, et al. Inhibition of curcumin on myeloid-derived suppressor cells is requisite for controlling lung cancer. Int Immunopharmacol. 2016 Oct;39:265-72.
25. Available at: https://www.cancer.org/latest-news/facts-and-figures-2018-rate-of-deaths-from-cancer-continues-decline.html. Accessed September 24, 2018.
26. Chen QY, Jiao DM, Yao QH, et al. Expression analysis of Cdc42 in lung cancer and modulation of its expression by curcumin in lung cancer cell lines. Int J Oncol. 2012 May;40(5):1561-8.
27. Xu X, Zhu Y. Curcumin inhibits human non-small cell lung cancer xenografts by targeting STAT3 pathway. Am J Transl Res. 2017;9(8):3633-41.
28. Lelli D, Pedone C, Majeed M, et al. Curcumin and Lung Cancer: the Role of microRNAs. Curr Pharm Des. 2017;23(23):3440-4.
29. Liu WL, Chang JM, Chong IW, et al. Curcumin Inhibits LIN-28A through the Activation of miRNA-98 in the Lung Cancer Cell Line A549. Molecules. 2017 Jun 3;22(6).
30. Zhan JW, Jiao DM, Wang Y, et al. Integrated microRNA and gene expression profiling reveals the crucial miRNAs in curcumin anti-lung cancer cell invasion. Thorac Cancer. 2017 Sep;8(5):461-70.
31. Chadalapaka G, Jutooru I, Chintharlapalli S, et al. Curcumin decreases specificity protein expression in bladder cancer cells. Cancer Res. 2008 Jul 1;68(13):5345-54.
32. Kelany ME, Hakami TM, Omar AH. Curcumin improves the metabolic syndrome in high-fructose-diet-fed rats: role of TNF-alpha, NF-kappaB, and oxidative stress. Can J Physiol Pharmacol. 2017 Feb;95(2):140-50.