The Powerful CancerCure Locked Inside Your Cells!

You might be familiar with coenzyme Q10—commonly called CoQ10—as a heart-saving miracle. This nutrient provides the fuel your heart needs to maintain its steady rhythm of 100,000 beats per day.

But recent clinical studies suggest this heart helper could be an outstanding cancer fighting nutrient too!

By Lee Euler / May 5, 2010

 According to the National Cancer Institute¹, coenzymes such as CoQ10 act as helpers to enzymes. The enzymes in your body speed up the pace of the natural chemical reactions in your cells.

You probably know that I consider enzymes to be sorely lacking in most folks’ nutritional supplement regimens. Enzyme deficiency is one of the biggest overlooked nutritional problems in American, in my opinion.

In fact, I wrote a report called “The Missing Ingredient for Good Health” to highlight the critical role of enzymes in fighting cancer… improving digestion… clobbering pain… and boosting circulation—just to name a few benefits!

Enzymes are essential to alternative cancer therapy. You’ll find that most alternative cancer doctors recommend either enzyme supplements or diets naturally rich in enzymes.

And as an enzyme’s helper—CoQ10 provides the boost that every cell in your body needs to function properly.

Continued below…

The only type of CoQ10 you should take now Find out how this Japanese breakthrough helps you achieve higher levels of CoQ10 in your bloodstream, more energy and even better antioxidant protection. This is the most bioavailable form of CoQ10 ever. It supports every part of your body. . . 1) Normalizes blood pressure 2) Supports better blood sugar levels 3) Restores healthy gums 4) Repairs damaged cells and even your DNA 5) Promotes healthy weight control 6) Even fosters younger looking skin! And if you or someone you love is taking a statin drug, this supplement breakthrough is critical for you. Statin drugs dangerously deplete your body’s natural CoQ10 levels. Click here to find out more about this amazing breakthrough.

Studies Prove CoQ10 Is A Superior Cancer Fighter

Interest in CoQ10 as a possible cancer treatment dates back to 1961. Researchers found that some cancer patients had lower than normal amounts of CoQ10 in their blood.

In fact, low blood levels of CoQ10 have been found in patients with various cancers including cancers of the breast, lung, prostate, pancreas, colon, kidney, and head and neck.

Some preclinical (animal) studies showed three beneficial effects of CoQ10 in treating cancer, namely:

1) CoQ10 boosted the immune system to help fight cancer and other diseases…

2) CoQ10 helped prevent heart damage in animals taking the anti-cancer drug doxorubicin…

3) CoQ10 analogs may have stopped cancer cells from growing…

Recent clinical studies also suggest that CoQ10 shows promise for treating cancer. Biochemical and Biophysical Research Communications reported on one 48-year-old man with heart failure who was diagnosed with inoperable lung cancer in 1977.

After receiving CoQ10 treatments, this man had no signs of either heart failure or cancer symptoms while taking CoQ10 for 17 years!

In 1993, Danish cancer specialist Dr. Knud Lockwood used CoQ10 as part of his recommended treatment for 32 breast cancer patients. Lockwood’s results were impressive—six of his 32 patients showed partial tumor remission!

In October 1993, a 74-year-old woman had a small tumor removed from her right breast. Encouraged by the results of Lockwood’s study, she refused a second operation to remove more growths and opted for 300 mg of CoQ10 daily.

Three months later, an examination and mammography revealed no evidence of the tumor or metastases! (http://www.ralphmoss.com/html/coQ10.shtml)

In most of these observed cases of heart disease and cancer, patients typically suffered from a CoQ10 deficiency.

I certainly wouldn’t recommend CoQ10 all by itself as a cancer “cure,” but it seems to me it would be a valuable addition to whatever else you’re doing. Boosting your CoQ10 intake could prove to be a safe and effective way to fight dangerous free radicals… protect yourself from cancer… and re-energize your entire body!

CoQ10 Helps Energize and Protect Your Cells!

CoQ10 works inside the cell mitochondria—the “energy factories” of your cells—to help transform foods you eat into the energy molecule ATP. ATP is then directed to areas of the cell where energy-consuming activities are taking place.

In addition to helping enzymes do their job—CoQ10 also doubles as a free radical fighter!

Free radicals form in the mitochondria when oxygen interacts with certain molecules. These harmful scavengers can destroy body cells if left unchecked.

In its role as an antioxidant, CoQ10 helps protect your cells from free-radical damage. These dangerous molecules even cause damage to your DNA, which some researchers have linked to the development of certain cancers.

You’ll probably NEVER hear anyone at the National Cancer Institute call CoQ10 a ‘cancer cure’! But the government agency does admit that CoQ10 is a powerful antioxidant that can help protect your body from cancer.

It is also commonly used as adjuvant therapy—that is, treatment given after a primary therapy to increase the chances of a cure.

Of course, the mainstream medical establishment insists on pushing their chemotherapy cocktails and surgeries first.

But the results of some animal and human studies show CoQ10 could be effective at treating cancers without resorting to popular ‘slash, poison, and burn’ remedies!

Try Nature’s Anti-Cancer Nutrient!

You may be surprised to know that CoQ10 has been listed as an essential nutrient in the Physicians’ Desk Reference for years. Also, the safety and effectiveness of oral supplements have been proven through numerous clinical studies.

CoQ10 is a natural substance found in most plant and animal cells. Some foods rich in the nutrient include:

• Anchovies
• Beef heart
• Broccoli
• Mackerel
• Nuts
• Salmon
• Sardines
• Spinach

These foods contain some of the CoQ10 you need to power up and protect your cells. However, many of the foods you eat may not contain enough CoQ10 to provide protection from cancerous growths.

Plus, aging decreases your body’s production of CoQ10. But you can find CoQ10 supplements at many nutritional supplement stores and websites. You’d be wise to take advantage of the protective benefits.

Is one CoQ10 supplement just like another?

There are a lot of choices available. Nearly every supplement brand has its own CoQ10. Is there any difference among them? As it turns out, there is.

95 percent of the CoQ10 in your blood is in a form called ubiquinol. But the form of CoQ10 found in nearly all supplements is ubiquinone. Your body has to convert the ubiquinone in the capsules into ubiquinol, and it’s not very efficient about doing that.

The reason supplement makers market the “inferior” form is that it doesn’t spoil as easily. The more potent form — ubiquinol — is very reactive with oxygen and oxidizes when exposed to air. In fact, the form found in the pills — ubiquinone — IS the oxidized form of CoQ10. You could say the CoQ10 in pills is the “spoiled” form!

But I’ve got good news: researchers have found a way to stabilize ubiquinol so it doesn’t oxidize, and you can now obtain this superior form of CoQ10 in supplements. Animal studies and some individual case studies in humans have confirmed that the superior form really is more effective.

It’s more expensive, but to get equivalent results with the cheaper form you need to take large doses. I’ve heard of heart patients taking up to 3,000 mg. a day of the cheaper form to get the desired results. The conversion factor I’ve seen is that 150 mg. of the superior form packs the same punch as 1200 mg. of the cheaper form. That’s a huge difference. 1200 mg. of the cheaper form won’t be CHEAP, I can tell you that — even if you buy it from one of the deep discounters.

One of our valued sponsors, Dr.Robert Rowen, offers ubiquinol in a formula that contains other valuable nutrients, and you can click here to see what he has to say. It’s a powerful formula. You may find yourself with more energy than you want! You can get ubiquinol elsewhere, if you choose to shop around. It’s your call.

For what it’s worth, if you buy from Dr. Rowen you’ll be helping to support his work as an alternative doctor as well as our efforts to tell people about alternative cancer treatments. He’s doing the Lord’s work in trying to break the medical monopoly in this country.

I realize some people think everything on the Internet should be free, but I can assure you that putting out this newsletter every week and creating our cancer reports is NOT free, so I deeply appreciate your help.

Either way, I hope you’re taking CoQ10 in some form every day!

https://www.cancerdefeated.com/the-powerful-cancercure-locked-inside-your-cellsrowen-coq10/

Higher cholesterol is associated with longer life

There are two types of cholesterol: High-density and low-density lipoprotein, aka HDL and LDL, but how they affect your heart and your longevity is the subject of ongoing controversy. For years, you've been told that high cholesterol levels were 'ba...

Analysis by Dr. Joseph MercolaFact Checked

September 02, 2019

STORY AT-A-GLANCE

• Cholesterol, the soft, waxy substance found in every cell in your body, is used to produce several of your body’s vital functions, including hormones and vitamin D. High cholesterol is now recognized for its link to longer life
• Faulty science says your total cholesterol is not a gauge of your heart disease risk, because when your levels are measured, elevated levels called triglycerides are also taken into account
• Found in every cell of your body, cholesterol is used to produce several of your body’s vital functions, including those that involve hormones and vitamin D
• Studies show a contrary or inverse link between all-cause mortality and total cholesterol levels; in other words, mortality is highest when your cholesterol is low, without exception
• Considering the false information on cholesterol, continued prescriptions for statins, and the potentially devastating side effects they cause, it’s clear why researchers are calling for a paradigm shift in the way cholesterol is treated

For those who aren’t sure the conventional health community is wholly in support of individual health when it comes to cholesterol levels — which in some cases still adheres to the story line that too much cholesterol increases the risk of heart disease — you’re right to be concerned.

Taking a global view of what cholesterol is and how it affects your body is a smarter way to approach it than the simple “cholesterol kills” narrative that’s been the drum beat for so many years.

Cholesterol, the soft, waxy substance found in every cell in your body, is used to produce several of your body’s vital functions, including those that involve hormones and vitamin D. About 75% of it is made by your liver and the remainder is derived from the foods you eat; 25% is in your brain.

There are two types: High-density is the first. It is also known as HDL, or the “good” kind that keeps cholesterol away from your arteries and removes it from your arteries. The second type is low density lipoprotein, or LDL. LDL is the “bad” kind that can build up in your arteries, form plaque that narrows your arteries and form a clot. This can then make its way to your heart or brain and cause either a heart attack or stroke.

The American Heart Association (AHA)1 now recommends that you balance your levels at about 150 (milligrams per deciliter) (mg/dL). According to old, unfounded science, your total cholesterol — the sum of all the cholesterol in your body — is not a gauge of your heart disease risk. When your levels are measured, elevated levels of triglycerides are also taken into account. According to Børge Nordestgaard, from the University of Copenhagen and Copenhagen University Hospital:

“So far, both cardiologists and [physicians] have focused mostly on reducing LDL cholesterol, but in the future, the focus will also be on reducing triglycerides and remnant cholesterol.”2

Does it matter what kills you?

In a commentary posted on Mission.org, a rhetorical question is presented: With all the ways you can die, does it really matter what kills you? On one hand, “If you’re dead, you’re dead, no matter from what,” so it seems silly to “focus on changing something that lowers the risk of death from one cause only to raise that risk from another.”3

It’s an astute observation when you read studies showing conclusively that cholesterol has very little to do with heart disease. Even more importantly, cholesterol is crucial for your health. If it’s too low, then eventually, your hormones, disease risk, cell signaling pathways, and yes, your heart, will suffer. In fact, new research shows that a too-low LDL level could put you at higher risk for a stroke.4

The Dietary Guidelines Advisory Committee (DGAC), which reviews the Dietary Guidelines for Americans every five years, investigated the issue. The 2015-2020 guidelines noted:5

“While adequate evidence is not available for a quantitative limit for dietary cholesterol in the 2015-2020 Dietary Guidelines, cholesterol is still important to consider when building a healthy eating style. In fact, the Dietary Guidelines states that people should eat as little dietary cholesterol as possible.”

Yet, even though that’s what it says on the government’s ChooseMyPlate public website, it’s obvious the committee at some point “flipped” its narrative entirely by acknowledging at a Dietary Guidelines Advisory Committee meeting that “cholesterol is not considered a nutrient of concern for overconsumption.”6

It’s no wonder people are confused about where cholesterol fits in your diet, when they send one message to the public and a completely different one to their own group at an advisory committee meeting.

Interestingly, that statement was made nearly five years ago, but information on the importance of cholesterol is nearly two decades old.7 As the Honolulu Heart Program study published in The Lancet in 2001 states:

"Our data accord with previous findings of increased mortality in elderly people with low serum cholesterol, and show that long-term persistence of low cholesterol concentration actually increases risk of death."

The narrative challenges those who insist that eating foods that contain fat of any kind — saturated fats and trans fats alike — are harmful. Many who make this claim also maintain that “Saturated fat is a bad fat because it raises your LDL level more than anything else in your diet.”8 However, as explained in the Mission.org article:

“While total cholesterol is a poor if not utterly worthless risk marker for heart disease, doctors have focused on it to the exclusion of how it might affect other causes of death. It does you little good to save yourself from heart disease if it means that you increase your risk of death from cancer. All-cause mortality — death from anything — is the most appropriate measure to use when looking at risk factors.”9

“Death from anything” may be a clearer term for the one so often used in clinical settings: All-cause mortality. Either way, as quoted by the following study, that’s what the latest research says is the best measure for the factors that increase the risk of what eventually and most likely will take someone’s life.

When it comes to your risk of death from heart disease, there’s much more evidence that inflammation is at the bottom of heart disease rather than high cholesterol, just as it is for a number of other serious diseases. If you want to find what might help you live longer, that’s the premise of a lengthy Japanese study published in the Annals of Nutrition & Metabolism. As the featured study, it notes that regardless of someone’s age, people with higher cholesterol live longer:

“Overall, an inverse trend is found between all-cause mortality and total (or low density lipoprotein [LDL]) cholesterol levels: mortality is highest in the lowest cholesterol group without exception. If limited to elderly people, this trend is universal. As discussed in Section 2, elderly people with the highest cholesterol levels have the highest survival rates irrespective of where they live in the world …

Based on data from Japan, we propose a new direction in the use of cholesterol medications for global health promotion; namely, recognizing that cholesterol is a negative risk factor for all-cause mortality and re-examining our use of cholesterol medications accordingly.”10

After showing that people of all ages with higher cholesterol levels live longer in Japan, in support of these conclusions, similar conclusions were made by a study based in the Netherlands and published in BMJ in 2016.11

The study adjusted for several heart risk factors like smoking, high blood pressure and a history of diabetes melllitus. Participants were placed in groups depending on whether their cholesterol levels were low, medium or high, and those with the highest cholesterol levels were found to have the lowest death rates.

The title of the BMJ review reveals the outcome — there was a “lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly.”12

In short, older people with high LDL cholesterol generally live just as long as — and may even outlive — people with low LDL, which begs the question of how the current cholesterol theory was arrived at in the first place. At the very least, the study authors imply the cholesterol question should at least be re-evaluated. It concluded:

“Our review provides the basis for more research about the cause of atherosclerosis and (cardiovascular disease) and also for a re-evaluation of the guidelines for cardiovascular prevention, in particular because the benefits from statin treatment have been exaggerated.”13

One of the first studies published with information suggesting that high cholesterol is not as heart-damaging as once thought was the Honolulu Heart Program,14 offering further evidence that higher cholesterol levels may be heart protective. The authors concluded by questioning whether there is “scientific justification for attempts to lower cholesterol to concentrations below 4-65 mmol/L in elderly people,” adding that “prudence dictates a more conservative approach in this age group.”15

Can high cholesterol really be heart protective?

Perhaps the question that would get to the point quicker is to ask why the Japanese study infers that people with low versus high cholesterol die sooner? The Mission offers referenced studies that show a few factors that could be weighing in:

•Cholesterol may protect against infections and atherosclerosis, as “the many observations that conflict with the LDL receptor hypothesis, may be explained by the idea that high serum cholesterol and/or high LDL is protective against infection and atherosclerosis.”16

•Cholesterol may protect against cancer, although in previous cases where low cholesterol was linked to cancer, exclusions were made to tip the scales, such as excluding possible prior drug treatment, namely clofibrate, a popular cholesterol-lowering drug before statins, by subjects — leaving the question open as to whether it was the low cholesterol that caused the cancer, or the drug treatments that contributed to it.17

•Low cholesterol (180 mg/dL and lower) and violence in psychiatric patients have been linked. When patients in a long-term psychiatric hospital with a history of seclusion or restraints were compared with other patients, there was a “highly significant and strong association between lower cholesterol levels and violent behavior.” The authors did caution, however, that cholesterol levels should not be used to predict violence.18

•There’s also an association between low cholesterol and suicide dating back more than a decade, as researchers found that people in the lowest quartile of cholesterol concentration had more than six times the suicide rates than those in the highest quartile.19

It should be noted that at least one study in Japan20 determined that high cholesterol and suicide were connected.

But, in fact, numerous recent studies have corroborated the earlier ones connecting low cholesterol to suicide,21 with various findings: For example, one found that low triglycerides, reduced BMI and waist circumference, specifically, but not total cholesterol, were connected to a higher risk of suicide;22 while a 2019 study23 found that “low cholesterol is associated with aggression in suicide attempters.”

So what about statins?

Significantly, the authors of an Annals of Nutrition & Metabolism study didn’t hold back when drawing conclusions regarding why the cholesterol conundrum has gone on so long when the evidence is so clear: “For the side defending this so-called cholesterol theory, the amount of money at stake is too much to lose the fight.”24

The Annals of Nutrition & Metabolism study’s introduction mentions a medical practitioner who advocated statins to his patients to drive down cholesterol until he read the Scandinavian Simvastatin Survival Study,25 in which 4,444 patients with different types of heart disease were given simvastatin — which is touted to be a safe, long-term treatment to improve survival in cardiovascular heart disease patients.

As it turns out, the claim that high cholesterol causes heart disease and death is incorrect; it is, in fact, the opposite. Three reviews26,27,28 supporting the cholesterol hypothesis were found to contain altered data to support their conclusions, according to Expert Review of Clinical Pharmacology, in which it is noted that:

“Our search for falsifications of the cholesterol hypothesis confirms that … the conclusions of the authors of the three reviews are based on misleading statistics, exclusion of unsuccessful trials and by ignoring numerous contradictory observations.”29

Drugs.com30 contributors state that the 35 million people on statins often experience myriad side effects. Liver damage, for instance, is said to be “rare,” implying that ongoing liver tests while taking statins likely aren’t necessary. Some doctors, however, say you’ll need a baseline liver function test beforehand. The most common side effects of statins are:

• Headaches, muscle pain, lower back or side pain
• Nasal congestion or stuffiness, or a runny nose
• Difficulty sleeping
• Constipation
• Hoarseness

And, just in case you needed another source to implicate statins’ role in psychiatric problems, an April 2018 study found that lowering cholesterol levels in men could bring about changes in nerve cell membranes and behavior in men:

“Men seem to be more sensitive to low cholesterol levels as the association between low cholesterol levels and aggression is found mostly in men,” the authors said.” … “Lowering cholesterol levels with statins brings about several changes in the serotonergic system, nerve cell membrane microviscosity and behaviour, and needs to be done with precaution in susceptible individuals.

Cholesterol levels could serve as a biological risk marker for violence and suicidal tendencies in psychiatric patients with depression and schizophrenia.”

More studies and reviews on statin use

Rather than pointing patients in the direction of finding dietary solutions, including eating both the whites and the yolks when having eggs, and ditching processed vegetable oils in favor of healthy cooking oils like coconut oil, olive oil and avocado oil, Harvard Health Medical School recently updated an article on how to “manage” muscle pain from taking statins, perpetuating the cholesterol myth. They stated:

“If you’re not taking a statin now, you may well be soon. These medications are commonly prescribed to lower ‘bad’ LDL cholesterol and have been shown to reduce the risk of heart attack, stroke, and death.

They are routinely recommended for people who have cardiovascular disease and for many people ages 40 to 75 who don't have cardiovascular disease but have at least one risk factor (high blood pressure, high cholesterol, diabetes, or smoking) and a 7.5% or greater risk of a stroke or heart attack in the next decade.

Moreover, recent research indicates that they may benefit high risk individuals over age 75 as well … Taking a statin may give you some assurance that you're doing all you can to avoid heart attack and stroke …”31

Exercising, losing weight, adopting healthy eating habits and including vitamin D and coenzyme Q10 (CoQ10) supplements in your diet are all good strategies for maintaining your cholesterol levels. Unfortunately, the common suggestion for alleviating muscle pain due to statin use is more of the same — just try taking a lower dose or switching to another statin prescription.32

As if that weren’t enough, experts say statins may impair your memory and cause amnesia, a possibility real enough to call for a warning on the labels of prescriptions.33 Statin use may precipitate a higher risk of developing cataracts34 and it “significantly” increases the likelihood of raising the fasting glucose levels of non-diabetics, as well as inducing high blood sugar.35

In another review of the adverse effects of statins, it was noted that “an array of additional risk factors for statin AEs [adverse events] are those that amplify (or reflect) mitochondrial or metabolic vulnerability, such as metabolic syndrome factors, thyroid disease, and genetic mutations linked to mitochondrial dysfunction.”36

Given the false information saying cholesterol is at fault for causing heart-related disease, the continued prescriptions being handed out to patients for statins, and the side effects they cause, it’s clear why, in their introduction, the authors of the Annals of Nutrition & Metabolism study stressed:37

“This, we believe, marks the starting point of a paradigm shift in not only how we understand the role cholesterol plays in health, but also how we provide cholesterol treatment … Our purpose in writing this supplementary issue is to help everyone understand the issue of cholesterol better than before, and we hope that we lay out the case for why a paradigm shift in cholesterol treatment is needed, and sooner rather than later.”

https://articles.mercola.com/sites/articles/archive/2019/09/02/higher-cholesterol-is-associated-with-longer-life.aspx

Preventing and Slowing Parkinson's Disease Progression

Parkinson's disease has burst into the public consciousness in recent years. It is a neurodegenerative (the breakdown of brain neurons) disease, resulting in a movement disorder.

Leader-Observer

06-25-19

Most notably, patients suffer from a collection of symptoms known by the mnemonic TRAP: tremors while resting, rigidity, inability/difficulty to move or slow movements and postural instability or balance issues.

It can also result in a masked face, one that has become expressionless, and potentially dementia, depending on the subtype.

The part of the brain most affected is the basal ganglia, and the prime culprit is dopamine deficiency in this brain region. Why not add back dopamine? Actually, this is the mainstay of medical treatment, but eventually the neurons themselves break down, and the medication becomes less effective.

Risk factors may include head trauma, reduced vitamin D, milk intake, well water, being overweight, high levels of dietary iron and migraine with aura in middle age.

Is there hope? Yes, in the form of medications and deep brain stimulatory surgery, but also with lifestyle modifications. Lifestyle factors include iron, vitamin D and CoQ10.

The role of iron

This heavy metal is potentially harmful for neurodegenerative diseases such as Parkinson's: it can cause oxidative damage.

In a small, yet well-designed, 12-month randomized controlled trial RC), researchers used a chelator to remove iron from the substantia nigra, a specific part of the brain where iron breakdown may be dysfunctional.

An iron chelator is a drug that removes the iron. Here, deferiprone DFP was used at a modest dose of 30 mg/kg/d. The chelator reduced the risk of disease progression significantly on the Unified Parkinson Disease Rating Scale (UPDRS).

Participants who were treated sooner had lower levels of iron compared to a group that used the chelator six months later. A specialized MRI was used to measure levels of iron in the brain.

The iron chelator does not affect, nor should it affect, systemic levels of iron, only those in the brain specifically focused on the substantia nigra region. It also may be recommended to consume foods that contain less iron.

CoQ10

CoQ10 is a coenzyme found in over-the-counter supplements, with doses from 100 to 300 mg. However, there is evidence that CoQ10 may be beneficial in Parkinson's at much higher doses.

In a 16-month trial with 80 patients, results showed that those given 1,200 mg of CoQ10 daily reduced the progression of the disease significantly based on UPDRS changes, compared to the placebo group. Lower doses did not show significant benefit.

Thus, there may be no downside to trying CoQ10 in those with Parkinson's disease.

Vitamin D: Good or bad?

In a forward-looking study, results show that vitamin D levels measured in the highest quartile reduced the risk of developing Parkinson's disease by 65 percent, compared to the lowest quartile.

This is quite impressive, especially since the highest quartile patients had vitamin D levels that were what we would qualify as insufficient, with blood levels of 20 ng/ml, while those in the lowest quartile had deficient blood levels of 10 ng/ml or less.

There were over 3,000 patients involved in this study with an age range of 50 to 79.

Many times we are deficient in vitamin D and have a disease, but replacing it does nothing to help the disease. Here, vitamin D may play dual roles of both reducing the risk of Parkinson's disease and slowing its progression.

In an RCT with 121 patients, results showed that 1,200 IU of vitamin D taken daily may have reduced the progression of Parkinson's disease significantly on the UPDRS compared to a placebo over a 12-month duration.

Also, this amount of vitamin D increased the blood levels by two times to 41.7 ng/ml.

Though medication with dopamine agonists is the gold standard for Parkinson's disease treatment, lifestyle modifications can have an impact on both prevention and treatment of this disease.

For further information, visit medicalcompassmd.com or consult your personal physician.

https://www.lifeextension.com/News/LefDailyNews?NewsID=29833&Section=Disease