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Showing posts with label PUFAs. Show all posts
Showing posts with label PUFAs. Show all posts

Sunday, 3 December 2017

American Heart Association President Suffers Heart Attack at 52

This is both a tragedy and an embarrassment for the Heart Association. Assuming the 52-year-old president was following the badly flawed AHA dietary guidelines, maybe this is the wake-up call they need to come clean with what the science really says for preventing heart attacks.

November 29, 2017

heart attack

Story at-a-glance

  • John Warner, cardiologist and president of the American Heart Association (AHA), recently suffered a heart attack in the middle of a health conference at the age of 52
  • In all likelihood, Warner followed AHA recommendations, many of which can actually worsen or cause heart disease
  • AHA supports ample grain consumption and recommends eating harmful fats such as canola, corn, soybean and sunflower oil, both of which are known to cause and/or contribute to cardiovascular problems
  • Good heart health starts with your diet — what you eat and when you eat. A powerful treatment for heart disease is to work your way up to an intermittent fasting schedule where you’re fasting for 20 hours a day
  • When you do eat, make sure you eat real food, and consider a cyclical ketogenic diet, high in healthy fats, low in net carbs with moderate protein. Once you’re comfortable with this intermittent fasting schedule, start doing a monthly water only fast, working your way up to multiple days
By Dr. Mercola
In the health paradox of the year, 52-year-old cardiologist John Warner, president of the American Heart Association (AHA), recently suffered a heart attack in the middle of a health conference.1,2 In a statement, the association reported Warner was in stable condition after having a stent placed to open a blocked artery. Part of Warner’s speech at the Scientific Sessions conference in Anaheim, California, centered around his own family’s struggle with heart disease.
"After my son was born and we were introducing him to his extended family, I realized something very disturbing: There were no old men on either side of my family. None. All the branches of our family tree cut short by cardiovascular disease," Warner said in his speech.3
“Together we can make sure old men and old women are regulars at family reunions, that people live long enough and healthy enough to enjoy walks and fishing trips with their grandchildren and maybe even their great-grandchildren. In other words, I look forward to a future where … children grow up surrounded by so many healthy, beloved, elderly relatives that they couldn't imagine life any other way.”
The AHA’s CEO, Nancy Brown, said in a statement:4 “John wanted to reinforce that this incident underscores the important message that he left us with in his presidential address … that much progress has been made, but much remains to be done.”

Many AHA Recommendations Worsen Heart Health

In all likelihood, Warner followed AHA recommendations, many of which are actually recipes for heart disease disaster. Of the foods scientifically proven to cause heart disease and clogged arteries, excess sugar and industrially processed omega-6 vegetable oils, found in nearly all processed foods, compete for space at the top the list. And what kinds of foods does the AHA recommend to protect your heart?
Not only does it support ample grain consumption, it also recommends eating harmful fats such as canola, corn, soybean and sunflower oil.5 “Blends or combinations of these oils, often sold under the name ‘vegetable oil,’ and cooking sprays made from these oils are also good choices,” the AHA says. Meanwhile, the association still insists saturated fats are to be avoided.
Just this past summer the AHA shocked health experts around the world by sending out a worldwide advisory6 saying saturated fats such as butter and coconut oil should be avoided to cut your risk of heart disease, and that replacing these fats with margarine and vegetable oil might cut your heart disease risk by as much as 30 percent. Overall, the AHA recommends limiting your daily saturated fat intake to 6 percent of daily calories or less.7
This is as backward as it gets, and if Warner was following this long-outdated advice, it’s no wonder he suffered a heart attack. In fact, it is to be expected. As noted by American science writer Gary Taubes in his extensive rebuttal to the AHA’s advisory,8 with this document, the AHA reveals its longstanding prejudice — and the method by which it reaches its flawed conclusions.
In short, the AHA simply excluded any and all contrary evidence. After this methodical cherry-picking, they were left with just four clinical trials published in the 1960s and early ‘70s — the eras when the low-fat myth was born and grew to take hold. The problem is nutritional science has made significant strides since then, and a number of significant studies have firmly disproven the hypothesis that saturated fat causes heart disease, finding no association whatsoever.
In related news, the AHA recently issued new guidelines on blood pressure,9 moving the goal post for heart health yet again. Now you’re considered hypertensive if your blood pressure is above 130 over 80. Previous guidelines started hypertension at 140 over 90. This means an estimated 30 million Americans will qualify for the designation of having high blood pressure, and of those, an estimated 1 in 5 are likely to receive the recommendation to take blood pressure medication.

Flawed Fat Recommendations Have Been Followed With Disastrous Consequences

Since the 1950s, when vegetable oils began being promoted over saturated fats like butter, Americans have dutifully followed this advice, dramatically increasing consumption of vegetable oil. Soybean oil, for example, has risen by 600 percent while butter, tallow and lard consumption has been halved. We’ve also dramatically increased sugar consumption, which has also been implicated as a primary contributor to heart disease and other chronic health problems.10
While following this advice, Americans have gotten fatter and sicker. Heart disease rates have not improved even though people have been following the AHA’s “heart healthy diet.” Common sense tells us if the AHA’s advice hasn’t worked in the last 65 years, it’s not likely to start working now. Modern research is just now starting to reveal what actually happens at the molecular level when you consume vegetable oil and margarine, and it’s not good.
For example, Dr. Sanjoy Ghosh,11 a biologist at the University of British Columbia, has shown your mitochondria cannot easily use polyunsaturated fatty acids (PUFAs) for fuel due to the fats’ unique molecular structure. Other researchers have shown the PUFA linoleic acid hinders mitochondrial function and can even cause cell death.12
PUFAs are also not readily stored in subcutaneous fat. Instead, PUFAs tend to get deposited in your liver, where they contribute to fatty liver disease, and in your arteries, where they contribute to atherosclerosis.
According to Frances Sladek,13 Ph.D., a toxicologist and professor of cell biology at UC Riverside, PUFAs behave like a toxin that builds up in tissues because your body cannot easily rid itself of it. Making matters worse, when vegetable oils like sunflower oil and corn oil are heated, cancer-causing chemicals like cyclic aldehydes are also produced.14
how the oils turn toxic
Source: The Telegraph November 7, 2015

Vegetable Oils Are Anything But Healthy

Other research confirms such findings by linking fried foods to an increased risk of death. For example, eating fried potatoes more than twice a week has been shown to double a person’s risk of death compared to never eating fried potatoes.15 Animal and human research has also found vegetable oils promote:
  • Obesity and fatty liver16
  • Lethargy and prediabetic symptoms17
  • Chronic pain/idiopathic pain syndromes (meaning pain with no discernible cause)18
  • Migraines19
  • Crohn’s disease and ulcerative colitis20
According to Dr. Cate Shanahan,21 a family physician and author of “Deep Nutrition: Why Your Genes Need Traditional Food,” the idea that PUFAs are healthier than saturated fats falls flat when you enter the field of biochemistry, because it’s “biochemically implausible.” In other words, the molecular structure of PUFA is such that it’s far more prone to react with oxygen, and these reactions disrupt cellular activity and cause inflammation.22 Oxidative stress and inflammation, in turn, are hallmarks not only of heart disease and heart attacks but of most chronic diseases.23,24
“[T]he folks at the AHA claim saturated fat is pro-inflammatory and causes arterial plaque and heart attacks — but there is no biochemically plausible explanation for their argument,” she told me in an emailed rebuttal to the AHA advisory.“Saturated fat is very stable, and will not react with oxygen the way PUFA fat does, not until the fundamental laws of the universe are altered.
Our bodies do need some PUFA fat, but we need it to come from food like walnuts and salmon or gently processed (as in cold pressed, unrefined) oils like flax and artisanal grapeseed, not from vegetable oils because these are refined, bleached and deodorized, and the PUFA fats are molecularly mangled into toxins our body cannot use.”

Open Letter to AHA President

In an open letter to AHA president Warner, Dr. William Davis, a New York cardiologist and author of The New York Times best seller “Wheat Belly, Lose the Wheat, Lose the Weight, and Find Your Path Back to Health,” writes, in part:
“If you ignore the nonsense that AHA policy dictates, you can absolutely gain control over cardiovascular risk. But you will NOT find the answers in any AHA policy. I learned these lessons practicing as an interventional cardiologist, then abandoning this ridiculous way of managing coronary disease to devote my efforts to early detection and prevention.
So, I thought I would articulate some of these thoughts in an open letter to Dr. Warner as he recovers from his procedure … Dr. Warner — … There are a number of reasons why someone like you — deeply-entrenched in the conventional world of heart disease management and what passes for prevention — highlights the miserable failure that the modern coronary care paradigm represents:
1) We are trapped by the outdated but profitable lipid hypothesis … 2) We know from abundant data that small oxidation- and glycation-prone LDL particles are highly atherogenic … are potent triggers of the inflammation cascade … and are triggered to abundant degrees in some genotypes upon consumption of the amylopectin A of grains …
[Y]es, the food that the American Heart Association advises to fill the diet with — and sugars … I am hoping that, now that this disease has touched you personally, your eyes will be opened to the corrupt and absurd policies of conventional coronary care and the American Heart Association.”

The Magic Pill Myth Needs to End

Davis goes on to note that heart disease is a multifactorial problem that cannot be solved with a pill.  
Thinking that a statin drug … [is] sufficient to prevent coronary events is absurd and overly-simplistic, like thinking that taking Aricept for dementia will stop the disease — of course, it does no such thing,” he writes, adding, “There are no drugs to ‘treat’ many of the contributors to coronary atherogenesis. But there are many non-drug strategies to identify, then correct, such causes.”
Nondrug prevention strategies suggested by Davis include:
Avoiding any and all dietary factors that provoke insulin resistance, glycation and formation of small, dense LDL particles. Importantly, this would include avoiding the harmful fats recommended by the AHA such as margarine and processed vegetable oils, and keeping your total daily fructose consumption below 25 grams per day.
Optimizing your vitamin D level.
Optimizing your omega-3 fat intake: An omega-3 index of 10 percent or greater is associated with “dramatic reduction in cardiovascular events,” Davis notes. Indeed, a 2010 analysis25 found that while diets higher in omega-6 fats (found in ample amounts in vegetable oils) and lower in omega-3s increased the risk of nonfatal myocardial infarction and death from heart disease by 13 percent; a mixed diet of both omega-3 and omega-6 fats reduced these risks by 22 percent.
Meanwhile, the AHA recommends higher intakes of omega-6, saying26 “Aggregate data from randomized trials, case-control and cohort studies, and long-term animal feeding experiments indicate that the consumption of at least 5 percent to 10 percent of energy from omega-6 PUFAs reduces the risk of coronary heart disease relative to lower intakes.
The data also suggest that higher intakes appear to be safe and may be even more beneficial.”This statement runs counter to a large body of research suggesting the converse — specifically, that reducing omega-6 fats and increasing omega-3 is better for your heart. 
Addressing your thyroid function.
Optimizing your gut microbiome to address dysbiosis caused by excess sugar, chlorinated and fluoridated water, and exposure to antibiotics, pesticides and common heartburn drugs.

Stent Placement No Better Than Placebo

Research also does not support the routine procedure of coronary artery angioplasty and stent placement. In fact, recent research suggests up to 50 percent of all stent placements may be unnecessary.27 Moreover, the effectiveness of this procedure is right on par with placebo. In a recent study published in The Lancet, researchers from Imperial College London investigated the difference between patients who had received a stent for stable angina and those who underwent a placebo intervention.28
In the short video above, lead author and interventional cardiologist Dr. Rasha Al-Lamee, describes the study and its results. Two hundred participants with severe single vessel blockage were recruited from five sites across the U.K.29 During the initial six weeks, all patients underwent an exercise test followed by intensive medical treatment.
At that point, they were randomly assigned to two groups. The first underwent a percutaneous intervention (PCI) during which coronary angioplasty was performed and a stent was placed. The second group also underwent a PCI procedure with an angiogram but without a balloon angioplasty or stent placement.30
For the following six weeks, neither the patient nor the physician knew if the patient received the stent. At the conclusion of the six weeks, patients again underwent an exercise test and were questioned about their symptoms. The researchers found both groups experienced nearly identical improvements in exercise tolerance and no difference in reported improvements of their symptoms.31 From the data, Al-Lamee commented:32
“Surprisingly, even though the stents improved blood supply, they didn’t provide more relief of symptoms compared to drug treatments, at least in this patient group. It seems that the link between opening a narrowing coronary artery and improving symptoms is not as simple as everyone had hoped.”

A New Way of Looking at Heart Disease

In this interview, Dr. Thomas Cowan, family physician, founding member of the Weston A. Price Foundation and author of “Human Heart, Cosmic Heart: A Doctor’s Quest to Understand, Treat and Prevent Cardiovascular Disease,” reveals how your heart and circulatory system works. This understanding may go a long way toward changing the way you understand heart disease.
He makes a strong case for heart disease being rooted in mitochondrial dysfunction and believes plaque formation alone cannot explain a heart attack."[Conventionally], it's all about the plaque," Cowan says"My point in the book is that it's NOT about the plaque." The conventional view is that your heart functions like a pump — a pressure propulsion system caused by the muscular contraction of the ventricles.
Cowan explains that your heart is actually better described as a hydraulic ram — a vortex-creating machine — where the primary mover of blood is the interaction occurring between the negatively charged vessel walls and the positively charged water in your blood. Importantly, the following three natural energies result in a separation of charges that improve blood flow:
1.Sunlight charges up your blood vessels, which increases the flow of blood. When the sun's rays penetrate your skin, it causes a massive increase of nitric oxide that acts as a vasodilator. As much as 60 percent of your blood can be shunted to the surface of your skin through the action of nitric oxide.
This helps absorb solar radiation, which then causes the water in your blood to capture the energy and become structured. This is a key component for a healthy heart. The ideal is to be exposed to the sun while grounding, meaning walking barefoot. This forms a biological circuit that makes it work even better.
2.Negative ions from the Earth, also known as earthing or grounding. This also charges up your blood vessels, creates a separation of charges, creates more positive ions and allows the blood to flow upward, against gravity. 
3.The field effect or touch from another living being, such as laying on of hands.

A Healthy Heart Is the Result of a Healthy Lifestyle

As noted by Cowan, "The best thing is to be, more or less, with shorts or naked on the beach, with the saltwater, which acts as an electrical conductor, holding hands with somebody you love. That's how you structure the water [in your blood vessels]." Sun exposure, grounding and skin-to-skin contact are three heart disease prevention strategies that, ideally, everyone should be doing, and it doesn't get a whole lot easier or less expensive than this.
That said, your heart health is really dependent on your diet — what you eat and when you eat. In my view, the best treatment for heart disease is to work your way up to an intermittent fasting schedule where you’re fasting for 20 hours a day. When you do eat, make sure you eat real food, and consider a cyclical ketogenic diet, high in healthy fats, low in net carbs with moderate protein.
Once you’re comfortable with this intermittent fasting schedule, start doing a monthly five-day water fast. This really is the most powerful metabolic intervention I know of, and I feel it’s one of the healthiest things I now do for my own health. Senescent cells, which have stopped replicating, play a distinct role in aging and disease. Once replication stops, these cells need to be removed from your body, or else they start clogging it up, causing severe inflammation and immune dysfunction.
Fasting very effectively gets rid of senescent cells — a process known as autophagy. Fasting also stimulates the production of stem cells, which help with regeneration and healing.
While a five-day fast may sound intimidating, if you’re used to 20-hour daily intermittent fasting for a month before starting your five-day fast, then the hunger that typically strikes on the second day of fasting is dramatically reduced and will typically not be at all bothersome. Fasting is also a powerful remedy for insulin resistance, which is a major underlying factor of heart disease.
Other important lifestyle strategies to protect your heart include getting enough CoQ10, getting regular exercise, making sure you get enough sleep (which is really important for mitochondrial health) and avoiding electromagnetic fields. To learn more about these, please read “CoQ10 — The No. 1 Supplement Recommended by Cardiologists,” “Here’s What Losing Sleep Does to Your Heart” and “The Real Dangers of Electronic Devices and EMFs.”
Last but not least, the following exercise, which requires only two to three minutes, three times a day, is a super-simple way to boost your heart health. It prompts your body to release nitric oxide, which will help relax your blood vessels and improve your blood pressure.

Now Available: ‘Fat for Fuel Ketogenic Cookbook’

I believe “Fat for Fuel” is one of the most important books I’ve ever written because it represents my worthy contribution to our understanding about preventing and healing disease. With all the information I packed into “Fat for Fuel,” it still didn’t provide the total picture. Although the book provides you with what changes are necessary and why to make them, you still need to know how to implement dietary changes and strategies.
That’s why I created a comprehensive companion tool, the “Fat for Fuel Ketogenic Cookbook: Recipes and Ketogenic Keys to Health from a World-Class Doctor and an Internationally Renowned Chef,” with celebrity chef Pete Evans. When you order today, you’ll receive three valuable bonuses:
  •  Bonus Chapter: Due to spacing considerations, this chapter was excluded from the book but can be yours today!
  •  Discount Coupon: A Mercola discount coupon good on your next order.
  •  Recipe Sneak Peek: An SMS exclusive with instant access to five recipes from the book.

Source: https://articles.mercola.com/sites/articles/archive/2017/11/29/aha-president-suffers-heart-attack.aspx

Thursday, 27 July 2017

Coconut Oil – Are You Coco-Nuts to Eat It?

Did you know that 50 percent of media headlines about medical studies are dead wrong? And that many of these headlines don’t accurately match the conclusions of the studies they cover?

Written by

Image result for coconut oil

That’s from a review published in the New England Journal of Medicine.
It makes me sad and furious at the same time that journalists don’t do their homework and create firestorms of confusion because of their negligent work.
That is exactly what happened when USA Today published their article, “Coconut Oil Isn’t Healthy. It’s Never Been Healthy.” Shame on you USA Today editors for doing such a sloppy job of journalism.
Why the American Heart Association Has Been and Still Is Wrong
First, there is not a single study showing that coconut oil causes heart disease. Not one. Second, the whole case against coconut oil is founded on a hypothesis that has been proven wrong. It’s the diet-heart hypothesis. Saturated fat raises LDL cholesterol. LDL cholesterol causes heart disease. Anything that raises LDL cholesterol is bad. Only problem is that the data does not support this hypothesis.
If you are geeky and want to read more where I cover the science in detail, read my blog “Fat: What I Got Wrong, What I Got Right.” But just like it took 150 years after Copernicus recognized that the earth revolves around the sun before it was finally accepted, it will take a while for the world to catch up with the false idea that low-fat and low-cholesterol diets won’t save us from heart disease. In fact, low-fat diets cause heart disease. I have covered all this in my last book, Eat Fat, Get Thin with hundreds of references.
The USA Today article was based on a review by the American Heart Association (AHA). They published a review of fat and heart disease. The AHA has been at the vanguard of bad advice for decades since they first hooked onto the “fat is bad and will kill you” meme. They told us to eat very low fat, low cholesterol diets and to eat tons of starchy carbs. By the way, the AHA gets huge funding from cereal makers that put the AHA seal of approval on sugary (including cereal makers) cereals because they are “fat free” despite containing 75 percent sugar. Except now the overwhelming amount of research has proved that idea to be dead wrong. In fact, their recommendations have killed millions of people (no joke) from heart disease and diabetes. That’s why the very conservative 2015 USDA Dietary Guidelines removed any upper limits on dietary fat and eliminated any restrictions on dietary cholesterol. In fact, after decades of telling us to avoid eggs and shrimp they said, “Cholesterol is not a nutrient of concern for overconsumption.”
If you are interested in the corruption of the AHA, its funding is supported by the Pharma industry, industrial food giants (including sugary cereal makers) and industrial vegetable oil manufacturers, then read this recent blog on Medium entitled, Is the American Heart Association a terrorist organization? I’m not calling the AHA a terrorist organization, I just want you to read the article and question who’s really pulling the strings.
Image result for coconut oil
How could this happen you might wonder? How could the scientists have gotten it so wrong? It all comes down to how we do the research. Most nutrition research is based on what we call observational studies. You follow a group of people for a long time, you ask them once a year what they ate last week and you see if patterns emerged. Good luck if they can remember. And people aren’t always honest – if they think butter is bad, then they will underreport consumption. Problem is those types of studies do not prove cause and effect, just correlation. If I did a study on women over 55-years of age who have sex, I could conclude that sex RARELY leads to pregnancy. Pretty meaningless. The populations studied that ate saturated fats also were coincidentally smokers, didn’t exercise and ate otherwise unhealthy diets. Those who actually ate well, exercised and didn’t smoke (i.e. lead healthier lifestyles) also didn’t eat saturated fats because they were told not to. Studies prove that it was the well-rounded healthy habits that saved them, not less saturated fats.
Why Saturated Fats Are Not Bad and Are Essential for Health
This is exactly why multiple recent studies have shown no link between saturated fat and heart disease. You can read most of the important ones from the references, below, in my book and in the Fat: What I Got Wrong, What I Got Right article. In fact, there have been very few cause and effect studies in nutrition – they are hard to do, take a long time and cost a lot. But two of the largest ones every published show that fat and saturated fat are not the problem. The first was the PREDIMED studywhere researchers gave 7,000 participants olive oil (which contains 20% saturated fat) or nuts vs. a low-fat diet. PERDIMED had to stop the study because the people who were following the low-fat diet were dying. 
Image result for coconut oil
The next study was completed over 40 years ago, but it wasn’t published because the results contradicted the prevailing dogma that saturated fat was bad and that LDL cholesterol caused heart disease. This was a study that could not be conducted today because it would be considered unethical. They fed 9,000 people in mental hospitals butter and saturated fats or corn oil (polyunsaturated vegetable oil, which the AHA report says we should eat more of). And, guess what? Those who ate the corn oil had more heart attacks and deaths, despite lowering their LDL cholesterol. Really? Yes, it’s true. In fact, for every 30-point drop in LDL the risk of heart attack went up 22 percent. To top it off a recent review of all the science on big bad butter looking at 6.5 million patient years of butter eating, researchers found that butter eaters had no increased risk of heart disease, but they did have decreased risk of type 2 diabetes. You read that right. Butter = lower risk of type 2 diabetes. And if you still need to be convinced, here’s a review of 17 meta-analyses(reviews of all the best and relevant research) showing no link between saturated fat and heart disease.
The whole idea that LDL cholesterol causes heart disease is the reason we have a multi-billion dollar statin industry. One study of over 130,000people who had heart attacks over 5 years showed that 75 percent had normal LDL and 50 percent had optimal LDL cholesterol. But only 10 percent had normal HDL or the protective cholesterol. Guess what raises HDL? Saturated fat. And coconut oil raises it the most of any saturated fat. And what lowers it? A low-fat, high-starch, and high-sugar diet. We need cholesterol and saturated fat for the health of every cell membrane, for your brain cells, your sex hormones and more. Cholesterol is not the cause of heart disease, it is the band-aid that tries to repair the arteries when damage occurs from a low-fat, high-starch, high sugar diet. This causes pre-diabetes and inflammation from a processed food diet, environmental toxins, a bad gut from a low fiber, processed diet or anything that causes inflammation.
Before we get off saturated fats, here’s one BIG warning. They ARE a problem if you eat them in the context of a high-sugar, high-starch, processed foods diet. It’s what I call sweet fat. Stay away. No bagels and butter. No donuts or French fries. Stick with butter and broccoli.
Why We Shouldn’t Be Mainlining Omega-6 Vegetable Oils
The other recommendation from the AHA other than dramatically lower saturated fat intake is to increase omega-6 refined vegetable oils. Bad idea. Yes, some studies show a lower risk of heart disease with a higher intake of omega-6 oils (otherwise known as polyunsaturated fats or PUFAs which include soy, corn, safflower, canola oils). Again I cover this in detail in Eat Fat, Get Thin, but the take-home is this: If you eat saturated fat in the context of the typical American diet, it will cause problems and the people in omega-6 studies were eating exactly that. And to make it more confusing, the studies looking at PUFAs included both omega-3 (super protective) and omega-6 fats. The omega-3s made the omega-6 cousins look better. When you look at just the studies of the effects of omega-6 fats, you find actually increase the risk of heart attacks. And it is just common sense and evolutionary sense – our intake of these refined, heat processed, hexane treated, chemically deodorized oils (never consumed before in human history) have increase more than 1000 times (that’s 100,000 percent) in the last 100 years. Saturated fats make your cholesterol molecules stable and less likely to oxidize or go rancid. It is oxidized cholesterol that causes heart attacks. The PUFA’s are unstable and easily oxidized so when they are incorporated into cholesterol they make it unstable and go rancid – which is bad for your heart.
Is Coconut Oil Healthy?
We have had a coconut craze. What’s the deal? Broccoli is healthy but if that’s all you ate you would get sick. Coconut oil is healthy but only as part of an overall healthy diet not as the main course. Coconut oil has been consumed by populations in the South Pacific for thousands of years without ill effect. It has so many health benefits. You can read more in my article Is Coconut Oil Bad for My Cholesterol. But here’s the short list of benefits. It raises HDL, the good cholesterol. It improves the quality and size and type of cholesterol. It lowers the total cholesterol to HDL ratio – a far better predictor of heart disease than LDL. And cultures with 60 percent of their diet as coconut oil have no heart disease. It also contains a unique type of saturated fat called MCT oil that boosts metabolism, reverses insulin resistance, and improves cognitive function. Coconut oil is also anti-fungal and anti-microbial and it contains lauric acid that is great for immune function. The only other good source of lauric acid is breast milk, which contains 24 percent saturated fat – far higher than the 6 percent the AHA recommends.
Who would you trust, nature/God or the American Heart Association? So, I am sorry you have to be buffeted about by bad conclusions from insufficient outdated science and bad journalism. But hopefully reading this helps, and if you are so inclined check out the references below, my other blogs on this topic, and my book so you can come to your own conclusions. 
Wishing you health and happiness,
Mark Hyman, MD

References
Appel, L.J., Sacks, F.M., Carey, V.J., Obarzanek, E. Swain, J.F., Miller, E.R. 3rd,OmniHeart Collaborative Research Group. (2005). Effects of protein, monounsaturated fat, and carbohydrate intake on blood pressure and serum lipids: results of the OmniHeart randomized trial. Journal of the American Medical Association, 294(19):2455-2464.
Assunção, M.L., Ferreira, H.S., dos Santos, A.F., Cabral, C.R., & Florêncio, T.M.M.T. (2009). Effects of dietary coconut oil on the biochemical and anthropometric profiles of women presenting abdominal obesity. Lipids, 44(7), 593-601.
Barberger-Gateua, P., Samieri, C., Feart, C., & Plourde, M. (2012). Dietary omega 3 polyunsaturated fatty acids and Alzheimer’s disease: interaction with apolipoprotein E genotype. Current Alzheimer’s Research, 8(5), 479-491.
Calder, P.C. (1998). Dietary fatty acids and the immune system. Nutritional Reviews, II, S70-S83.
Cardoso et al. (2015). A coconut extra virgin oil-rich diet increases HDL cholesterol and decreases waist circumference and body mass in coronary artery disease patients. Nutrition Hospitals, 32(5), 2144-2152. doi: 10.3305/nh.2015.32.5.9642.
Crawford, M.A., Broadhurst, C.L., Guest, M., Nagar, A., Wang, Y., Ghebremeskel, K., & Schmidt, W. (2013). A quantum theory for the irreplaceable role of docosahexaenoic acid in neural cell signaling throughout evolution. Prostaglandins Leukotrienes and Essential Fatty Acids, 88(1), 5-13.
Daley, C. A., Abbott, A., Doyle, P. S., Nader, G. A., & Larson, S. (2010). A review of fatty acid profiles and antioxidant content in grass-fed and grain-fed beef. Nutrition Journal, 9(1), 10.
Das, U.N. (1994). Beneficial effect of eicosapentaenoic and docosahexaenoic acids in the management of systemic lupus erythematosus and its relationship to the cytokine network. Prostaglandins Leukotrienes and Essential Fatty Acids, 51(3), 207-213.
de Souza et al. (2015). Intake of saturated and trans unsaturated fatty acids and risk of all cause mortality, cardiovascular disease, and type 2 diabetes: a systematic review and meta-analysis of observational studies. British Medical Journal, 351.
Estruch, R., Ros, E., Salas-Salvado, J., Covas, M.I., Corella, D., Aros, F.,…PREDIMED Study Investigators. (2013). Primary prevention of cardiovascular disease with a Mediterranean diet. New England Journal of Medicine, 368(14), 1279-1290. doi: 10.1056/NEJMoa1200303
FAO/WHO. (2009). Fats and fatty acids in human nutrition. Proceedings of the Joint FAO/WHO Expert Consultation. November 10-14, 2008. Geneva, Switzerland. Annals of Nutrition and Metabolism, 55, 1-3.
Fernando, W.M.A.D.B., Martins, I.J., Goozee, K.G., Brennan, C.S., Jayasena, V., & Martins, R.N. (2015). The role of dietary coconut for the prevention and treatment of Alzheimer’s disease: potential mechanisms of action. British Journal of Nutrition, 1-14.
Intahphuak, S., Khonsung, P., & Panthong, A. (2010). Anti-inflammatory, analgesic, and antipyretic activities of virgin coconut oil. Pharmacological Biology, 48(2), 151-157.
Kalmijn, S., Feskens, E.J.M., & Kromhout, D. (1997). Polyunsaturated fatty acids, antioxidants, and cognitive function in very old men. American Journal of Epidemiology, 145, 33-41.
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